Irritable bowel syndrome (IBS) is distinguished by the frequent coexistence of digestive, mental, and somatic comorbidities, which contribute significantly to its severity, treatment refractoriness, increased health care utilization, and reduced quality of life.1 Despite incomplete understanding, the generation of dysfunction and symptoms in IBS has been linked to alterations in gut and brain immunity, intestinal microbiota, neuroenteroendocrine production, and motility.2,3
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